Pulmonary Surfactants

Key Points

  • Pulmonary surfactant is a phospholipid secreted by type-II alveolar cells that reduces surface tension in the lungs, preventing alveolar collapse during exhalation.
  • Preterm infants (<32 weeks’ gestation) do not produce adequate surfactant, resulting in respiratory distress syndrome (RDS).
  • Exogenous surfactant (beractant/Survanta) is collected from animal lungs and administered via endotracheal tube to preterm infants.
  • Dose: 4 mL/kg (100 mg phospholipids/kg) administered in four 1 mL/kg aliquots; up to 4 doses in first 48 hours, no more frequently than every 6 hours.
  • Monitor for oxygen desaturation and transient bradycardia during administration; withhold suctioning for 1 hour after each dose unless airway obstruction occurs.

Pathophysiology of Surfactant Deficiency

Pulmonary surfactant is a surface-active phospholipid produced by alveolar type-II (AT-II) epithelial cells. Its function is to decrease surface tension at the gas-liquid interface of the alveoli, preventing collapse during exhalation.

Development timeline: Lung-surfactant production begins at approximately 24 weeks’ gestation, but adequate amounts are not produced until at least 32 weeks’ gestation.

In preterm infants, surfactant deficiency causes:

  1. Alveolar collapse with each breath (atelectasis)
  2. Increased work of breathing — the infant must generate extreme pressure to reinflate collapsed alveoli
  3. Progressive hypoxia and hypercapnia
  4. Respiratory failure if untreated

Respiratory Distress Syndrome (RDS)

RDS (formerly called hyaline membrane disease) is the primary indication for surfactant therapy:

FeatureDetails
PopulationPreterm infants; higher risk with lower gestational age; white males in late preterm also at risk
SignsTachypnea, nasal flaring, expiratory grunting, multilevel retractions, cyanosis
Chest X-rayLow lung volumes, diffuse reticulogranular “ground glass” pattern, air bronchograms
CourseWorsens over first 2–3 days; improves as endogenous surfactant production begins

Drug: Beractant (Survanta)

Drug class: Exogenous pulmonary surfactant (bovine-derived)

Mechanism: Provides exogenous phospholipids to replace deficient surfactant, reducing alveolar surface tension and restoring functional residual capacity.

Indications:

  • Prevention and treatment of RDS in premature neonates
  • Lung immaturity in neonates at risk for RDS

Dosing

ParameterDetails
Dose4 mL/kg (100 mg phospholipids/kg)
First doseAs soon as possible after birth — preferably within 15 minutes
FrequencyUp to 4 doses within the first 48 hours of life; no more frequently than every 6 hours
MaximumUsually requires no more than every 12 hours unless surfactant is being inactivated by infection

Administration Technique

ETT Required

Beractant is administered exclusively via endotracheal tube. The infant must be intubated and stable prior to administration.

  1. Confirm infant is stable before proceeding
  2. Insert a 5 French end-hole catheter into the endotracheal tube
  3. Administer dose in four 1 mL/kg aliquots (quarter-doses)
  4. Each quarter-dose instilled over 2–3 seconds, followed by ≥30 seconds of manual ventilation or until infant is stable
  5. Each quarter-dose administered in a different position:
    • Slightly downward with head turned right
    • Slightly downward with head turned left
    • Slightly upward with head turned right
    • Slightly upward with head turned left
  6. Withhold suctioning for 1 hour after full dose — unless signs of significant airway obstruction occur

Nursing Assessment

NCLEX Focus

Surfactant is administered via ETT — nurses must monitor O₂ saturation and heart rate closely during each aliquot. Withhold suctioning for 1 hour post-dose. Improvement in oxygenation after surfactant = expected response, not a complication.

  • Assess infant’s respiratory status before and throughout administration: SpO₂, heart rate, breath sounds, work of breathing
  • Assess ventilator settings — as oxygenation improves after surfactant, ventilator support may need to be weaned
  • Monitor for diuresis — may occur as oxygenation improves
  • Confirm correct ETT placement before each dose

Adverse Effects and Monitoring

EffectTimingNursing Action
Oxygen desaturationDuring instillationPause, ventilate manually until stabilized
Transient bradycardiaDuring instillationPause, assess, ventilate; notify provider if persistent
Blood pressure alterationsDuring/after doseContinuous monitoring; report significant changes
Pulmonary hemorrhageRare, severeEmergent respiratory support; notify provider immediately
Patent ductus arteriosus (PDA)Post-treatmentAssess for bounding pulse, murmur; echocardiogram if suspected

Nursing Interventions

  • Warm refrigerated vial to room temperature by holding in hands for at least 8 minutes — do NOT microwave or shake
  • Confirm ETT patency and position before administration
  • Position infant per administration protocol (four positional aliquots)
  • Monitor SpO₂ and heart rate continuously during administration — pause and ventilate if desaturation or bradycardia occurs
  • Withhold airway suctioning for 1 hour after dose unless obstruction occurs
  • Anticipate progressive improvement in oxygen requirements — wean supplemental oxygen and ventilator support as directed as oxygenation improves
  • Educate parents: explain that the infant is receiving surfactant to replace what the premature lungs cannot yet produce on their own

Self-Check

  1. A preterm infant at 28 weeks’ gestation is intubated and receiving beractant. During the second quarter-dose, SpO₂ drops to 82%. What is the priority nursing action?
  2. Why must suctioning be withheld for 1 hour after surfactant administration?
  3. After beractant administration, the nurse notes improved SpO₂ and a decrease in FiO₂ requirements. Is this a normal response or a complication?