HIV and AIDS

Key Points

  • HIV (human immunodeficiency virus) progressively destroys CD4+ T-helper lymphocytes, impairing cell-mediated immunity.
  • AIDS (acquired immunodeficiency syndrome) is diagnosed when the CD4 count falls below 200 cells/µL or an AIDS-defining illness occurs.
  • Antiretroviral therapy (ART) suppresses viral replication, restores immune function, and prevents progression — but does not cure HIV.
  • Adherence is the highest-priority nursing concern — missed doses lead to drug resistance and treatment failure.
  • Standard precautions must be used with ALL patients regardless of HIV status.

Pathophysiology

HIV binds to CD4+ receptors and CCR5/CXCR4 co-receptors on T-helper lymphocytes. Reverse transcriptase converts viral RNA to DNA, which integrase incorporates into host cell DNA. The infected cell becomes a viral factory, producing new HIV particles that bud off and infect additional CD4 cells.

As CD4 cells are destroyed, cell-mediated immunity progressively deteriorates, leaving the patient vulnerable to opportunistic infections — pathogens that would not cause disease in immunocompetent individuals.

Stages of HIV Infection

StageCD4 CountCharacteristics
Stage 1: Acute HIV≥500 cells/µLFlu-like syndrome 2–4 weeks after exposure; high viral load; highly infectious
Stage 2: Chronic HIV200–499 cells/µLOften asymptomatic; slow viral replication continues; still transmissible
Stage 3: AIDS<200 cells/µLOpportunistic infections; wasting syndrome; high viral load; life-threatening

Transmission Routes

  • Sexual contact (most common): unprotected vaginal, anal, or oral sex
  • Blood-to-blood: sharing IV drug needles, contaminated blood products, needlestick injuries
  • Perinatal: transplacental, during childbirth, or via breast milk

HIV is not transmitted by casual contact (handshakes, hugging, shared food/water, coughing, sneezing).

Opportunistic Infections (AIDS-defining illnesses)

InfectionCD4 ThresholdDescription
Pneumocystis jirovecii pneumonia (PCP)<200 cells/µLMost common AIDS-defining infection; prophylaxis with TMP-SMX
Toxoplasma encephalitis<100 cells/µLBrain lesions; headache, altered consciousness
Cytomegalovirus (CMV) retinitis<50 cells/µLProgressive blindness if untreated
Mycobacterium avium complex (MAC)<50 cells/µLDisseminated infection; fever, night sweats, weight loss
Kaposi sarcomaAIDS-definingPurplish skin lesions; HHV-8 associated

Nursing Assessment

NCLEX Focus

Monitor CD4 count (immune reserve) and viral load (treatment effectiveness) at every visit. Undetectable viral load (<50 copies/mL) confirms ART effectiveness. CD4 <200 triggers prophylactic antibiotics for PCP.

  • Monitor CD4 count and viral load trends.
  • Assess for constitutional symptoms: fever, night sweats, weight loss, fatigue, lymphadenopathy.
  • Assess for opportunistic infection signs: oral candidiasis (thrush), herpes outbreaks, pneumonia symptoms, neurological changes.
  • Assess ART adherence: pill counts, refill history, patient self-report, identify barriers.
  • Assess for ART adverse effects: GI symptoms, hepatotoxicity, metabolic syndrome (PI-associated), peripheral neuropathy (older NRTIs).
  • Assess mental health: depression, anxiety, and stigma-related distress are common.
  • Assess social determinants: housing stability, food security, social support — all affect adherence.

Nursing Interventions

  • ART adherence counseling: Non-judgmental approach; explore barriers (cost, side effects, depression, schedule complexity); reinforce that consistent adherence achieves undetectable viral load.
  • Infection prevention: Educate on hand hygiene, avoiding undercooked foods, avoiding exposure to individuals with active infections; practice respiratory hygiene.
  • Standard precautions: Apply standard precautions with ALL patients regardless of HIV status — gown, gloves, mask, eye protection per exposure risk.
  • Transmission prevention education: Consistent condom use; U=U principle (Undetectable = Untransmittable — when viral load is undetectable, sexual transmission risk approaches zero); clean needle programs; PrEP for partners.
  • PrEP (pre-exposure prophylaxis): Offer to HIV-negative high-risk individuals — tenofovir/emtricitabine (Truvada); requires testing every 3 months.
  • PEP (post-exposure prophylaxis): Must start within 72 hours of potential exposure; ART regimen continued for 28 days.
  • Psychosocial support: Therapeutic communication; preserve dignity and confidentiality; connect to HIV support groups and mental health services.
  • Nutrition: Consult dietician; maintain adequate protein and caloric intake; address wasting syndrome.

Needlestick Exposure Protocol

Following a needlestick or mucocutaneous exposure to HIV+ blood: (1) Wash site immediately with soap and water; (2) Report to occupational health immediately; (3) Draw blood from source patient if possible; (4) Start post-exposure prophylaxis (PEP) within 72 hours — delay reduces effectiveness; (5) Continue PEP for 28 days.

Pharmacology

See antiretroviral-therapy for complete ART drug class overview (NRTIs, NNRTIs, PIs, INSTIs, fusion inhibitors, CCR5 antagonists).

First-Line Regimen ExampleDrug ClassesNotes
Bictegravir/TAF/emtricitabine (Biktarvy)INSTI + 2 NRTIsOnce-daily; high genetic barrier to resistance
Dolutegravir + 2 NRTIsINSTI backbonePreferred for most patients

Opportunistic infection prophylaxis:

CD4 ThresholdProphylaxis DrugTarget
<200 cells/µLTMP-SMX (Bactrim)PCP (Pneumocystis pneumonia)
<100 cells/µLTMP-SMX ± pyrimethamineToxoplasma encephalitis
<50 cells/µLAzithromycin or clarithromycinMAC (Mycobacterium avium complex)

Clinical Judgment Application

Clinical Scenario

A patient with HIV on ART for 2 years has a new CD4 count of 148 cells/µL (previously 380) and a viral load of 12,500 copies/mL (was undetectable). They admit to missing doses “frequently.”

  • Recognize Cues: Rising viral load, declining CD4, known adherence failure in patient on established ART.
  • Analyze Cues: Nonadherence has allowed viral replication and likely drug resistance development.
  • Prioritize Hypotheses: Drug resistance and AIDS-stage immunosuppression are the highest concerns.
  • Generate Solutions: Initiate genotypic resistance testing; reassess ART regimen; address adherence barriers; start PCP prophylaxis (CD4 <200); refer to social work for support services.
  • Take Action: Non-judgmental adherence counseling; coordinate with infectious disease specialist; begin TMP-SMX prophylaxis per provider order.
  • Evaluate Outcomes: Viral load becomes undetectable within 3–6 months with consistent adherence; CD4 recovers toward >200.

Self-Check

  1. What CD4 count defines the diagnosis of AIDS, and why does it mark a critical threshold?
  2. Within what time window must post-exposure prophylaxis (PEP) begin to be effective?
  3. What does “U=U” mean, and why is it important patient education for HIV transmission prevention?